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GLP-1 S (5mg) / Tesofensine (500mcg) x 30 Tablets

GLP-1 S (5mg) / Tesofensine (500mcg) x 30 Tablets

$605.00 USD
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GLP-1 S (5mg) / Tesofensine (500mcg) x 30 Tablets

$605.00 USD

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What is GLP-1 S (5mg) / Tesofensine (500mcg) x 30 Tablets?

What is GLP-1 S / Tesofensine?

GLP-1 S / Tesofensine is a research combination designed to explore the interaction between metabolic peptide regulation and central monoaminergic signaling. GLP-1 S is a synthetic analog related to GLP-1 receptor peptides and studied for its effects on metabolic and receptor pathways, while Tesofensine is a monoamine reuptake inhibitor analog studied for its role in appetite and energy control research. Together, they represent a complementary system that merges peptide signaling with neurotransmitter regulation, providing researchers with a model for studying cross-talk between peripheral metabolic peptides and central neurochemical pathways.

Chemical Structure of Reta GLP-1 S (5mg) / Tesofensine (500mcg) x 30 Tablets

What is the Chemical Structure of GLP-1 S / Tesofensine?

GLP-1 S is a long-chain synthetic peptide composed of approximately 30 amino acids, designed to emulate the active region of GLP-1 receptor agonists. Its molecular configuration includes modifications that enhance receptor affinity, increase peptide half-life, and support biochemical stability in vitro. Tesofensine is a tropane-based compound with the molecular formula C₁₇H₂₃NO and a molecular weight of 257.37 g/mol. It features a bicyclic tropane core with a tertiary amine that interacts with monoamine transporters, enabling strong affinity for dopamine, serotonin, and norepinephrine reuptake sites. When combined, the peptide component (GLP-1 S) and small-molecule component (Tesofensine) form a dual-modality formulation that bridges peptide receptor signaling with central neurotransmitter modulation in metabolic research models.

What Are the Effects of GLP-1 S (5mg) / Tesofensine (500mcg) x 30 Tablets

What Are the Effects of GLP-1 S / Tesofensine?

GLP-1 S has been studied for its activation of GLP-1 receptor pathways involved in peptide-mediated metabolic signaling, glucose regulation, and satiety responses. Research indicates it enhances receptor-driven cAMP signaling and energy utilization. Tesofensine has been studied for its inhibition of norepinephrine, dopamine, and serotonin reuptake, leading to increased synaptic neurotransmitter availability and central appetite regulation. When examined together, GLP-1 S and Tesofensine have demonstrated complementary effects in experimental models: GLP-1 S acts peripherally on metabolic peptides and receptor pathways, while Tesofensine acts centrally on neurotransmitter signaling, producing a synergistic influence on metabolic and neuroendocrine balance.